Answer :
The major site of conversion of angiotensin I to angiotensin II is the lungs. The mechanism causing tachycardia in the given scenario is mediated by arterial baroreceptors, which respond to low blood pressure by increasing heart rate.
Therefore the correct option is 2 and 5.
From the options given, the major site of conversion of angiotensin I to angiotensin II is the lungs. The enzyme responsible for this conversion is angiotensin-converting enzyme (ACE), which is produced in the lungs.
The neutral mechanism responsible for the development of tachycardia (increased heart rate) in this situation is mediated by the arterial baroreceptors. These receptors detect low blood pressure and signal the autonomic nervous system to increase heart rate to maintain adequate blood circulation.
The major site of conversion of angiotensin I to angiotensin II is option2. Lungs. The neural mechanism most likely responsible for the development of tachycardia in this man is immediately by which of the following receptors is option 3 Cutaneous thermoreceptors.
Option2. Lungs
Angiotensin-converting enzyme (ACE) is primarily found in the endothelial cells of the lungs. It converts angiotensin I, which is produced in the liver and released into the bloodstream, to angiotensin II. Angiotensin II is a potent vasoconstrictor and plays a central role in regulating blood pressure and fluid balance.
Option3.Cutaneous thermoreceptors
On a hot day, increased body temperature can lead to activation of cutaneous thermoreceptors in the skin. This activation triggers a sympathetic response, causing peripheral vasodilation and increased sweating to dissipate heat. In response to the peripheral vasodilation, there is a compensatory increase in heart rate (tachycardia) mediated by the baroreceptor reflex. Arterial baroreceptors play a role in regulating blood pressure, but they are not the primary receptors involved in this specific scenario of heat-induced tachycardia.
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